In the past few years, secondary abiotic as well as conventional NOD2 deficient IL-10−/− (NOD2−/− IL-10−/−) mice were used to unravel the role of the innate immune receptor nucleotide-oligonucleotide-domain 2 (NOD2) during campylobacteriosis and clearance of C. jejuni infection [51,142,143,144,145,146]. This evidence concerns the gene IL10 and campylobacteriosis.