CRY2 and Alzheimer disease: Our approach leads to the expression of Aβ-CRY2-mCh in only a small subset of neurons making analysis of AD hallmarks difficult, so we used transient expression of Aβ-CRY2-mCh driven by a ubiquitin promotor in 48 hr old embryos to quantify the effects of light-induced Aβ oligomerization on mitochondria health and metabolic deficits.