TGFB1 and renal fibrosis: In other models of renal fibrosis following non‐infectious insult, MSC‐like cell populations residing in the kidney and recruited from circulating precursors become activated myofibroblasts after local exposure to TGFβ1, subsequently depositing extracellular matrix components around the injured area that ultimately form a scar (Humphreys, 2018; Meng, Nikolic‐Paterson, & Lan, 2016).