To examine the possibility that TRIM32 regulates the growth of highly glycolytic tumor cells, we confirmed that overexpression of the activated platelet PDGF/VEGF receptor (Pvract) in dpp-expressing wing disc cells increased LDH-GFP expression and promoted tissue overgrowth (Figure 7B,D; Figure 7—figure supplement 1C; Wang et al., 2016). Here, TRIM32 is linked to neoplasm.