In Kallmann syndrome, anosmia/hyposmia is part of the clinical picture, and ANOS1, CHD7, FGFR1, PROK2, PROKR2, and SEMA3A variants were reported to be involved in isolated congenital anosmia (Alkelai et al. 2017). This evidence concerns the gene FGFR1 and Kallmann syndrome.