We further observed that miR‐155‐3p knock‐down in both A172 and U87 glioma cells increased endogenous Six1 levels and its downstream proteins, but no notable change was observed in the transcript levels (Figure 4D), thus indicating the inhibition of Six1 by miR‐155‐3p at the translational and not transcription level. The gene discussed is SIX1; the disease is central nervous system cancer.