Finally, we extended ST3GAL6-AS1 and ST3GAL6 expression analysis to a representative panel of human hematological tumors, including MM (n = 21), lymphoma (n = 6) and leukemia (n = 5) cell lines, and found that the expression levels of both ST3GAL6-AS1 and ST3GAL6 were significantly higher in human MM cell lines (HMCLs) as compared with other tumor types (Figure 4 and Figure S3). This evidence concerns the gene ST3GAL6 and Miyoshi myopathy.