FOXM1 and myocardial infarction: MSC-derived lncRNAs have been shown to support angiogenesis through the endothelial differentiation of MSCs: myocardial infarction-associated transcript (MIAT) targets miR200a and VEGF [85, 86]; maternally expressed gene 3 (MEG3) facilitates the ubiquitination and degradation of forkhead box protein M1 (FOXM1) reducing the angiogenic VEGF expression and promoting endothelial differentiation of MSCs [87].