Unfortunately, they did not further investigate the value of AKT3, NFATc2, or PPP3CA for the function of HSDL2, but we conducted rescue experiments confirming that AKT2 plasmid significantly reversed the suppression effects of siHSDL2 on lung adenocarcinoma cell proliferation, invasion, and migration. The gene discussed is HSDL2; the disease is lung adenocarcinoma.