However, a more recent study discovered increased levels of miR-125b induced by the overexpression of octamer-binding transcription factor 4 (OCT4) gene, which, in turn, reduced the expression of Bcl-2 homologous antagonist/killer (BAK1) protein, therefore decreasing the apoptosis of cervical cancer cells (Wang et al., 2013). This evidence concerns the gene BAK1 and cervical carcinoma.