Differential expression of antiinflammatory cytokines (IL-10, TGF-β, TLR4 and NF-κB) occurs in women with endometriosis, and this can promote survival, growth, invasion, differentiation, angiogenesis, and immune escape of the endometriotic lesions (Zhou et al., 2019). Here, NFKB1 is linked to endometriosis.