Differential expression of antiinflammatory cytokines (IL-10, TGF-β, TLR4 and NF-κB) occurs in women with endometriosis, and this can promote survival, growth, invasion, differentiation, angiogenesis, and immune escape of the endometriotic lesions (Zhou et al., 2019). The gene discussed is TLR4; the disease is endometriosis.