Our data showed that overexpression of Cpt1b significantly attenuated sh-ERRγ-increased sensitivity of Dox in HepG2/ADR cells (Figure 6H), and also sh-ERRγ-downregulated ATP levels (Figure 6I) and FAO rate (Figure 6J), suggesting that Cpt1b is involved in ERRγ-regulated FAO and chemoresistance of cancer cells. Here, CPT1B is linked to cancer.