We also report an increase in lung neutrophil counts in Gcnt1−/− mice, which may seem counterintuitive, as absence of Gcnt1 associates with a partial deficiency of selectin ligands.11 However, the recruitment of neutrophils to the lung may follow an atypical selectin-independent cascade,9,31 as shown during Streptococcus pneumonia infection.32 Also, Gcnt1 activity has been demonstrated to be dispensable for lung neutrophil recruitment upon LPS stimulation.33 We show that lung neutrophilia in Gcnt1−/− mice involved both hematopoietic and stromal compartments. Here, GCNT1 is linked to medical procedure.