Deficiency in the α1,3-fucosyltransferase Fut7 or Fut4/Fut7 double deficiency impaired T cell homing to the lymph nodes during Mtb experimental infection, but the traffic of effector T cells to the lung was normal.19 Both Fut7 deficient (−/−) and Fut4−/−/Fut7−/− mice were initially shown to resist normally to low dose aerosol infection,19 but a shorter survival of these mice upon infection was reported in a later study.20 The gene discussed is FUT7; the disease is infection.