Interestingly, it does not rely on differential expression of Cxcl5, a chemokine produced by epithelial cells that drives neutrophil recruitment, contributing to more severe forms of TB.34 In the same context, Gcnt1−/− neutrophils showed migratory profiles towards CXCL1, CXCL2 and CXCL5 similar to those of C57BL/6 neutrophils. This evidence concerns the gene CXCL2 and tuberculosis.