Several studies have provided evidence that the Janus kinase (JAK)/signal transducer and activator of transcription (STAT), platelet-derived growth factor (PDGF), NOTCH-1, nuclear factor (NF)-κB pathways in NKTCL were dysregulated (9, 13, 14). The gene discussed is SOAT1; the disease is extranodal nasal NK/T cell lymphoma.