Mlynarski et al. (2005) showed that the CCR5-Δ32 mutation increased the risk of kidney disease in men with type 1 diabetes; however, this outcome is contrary to that of Prasad et al. (2007) who found that CCR5-Δ32 was not related to nephropathic type 2 diabetes patients. Skrzypkowska et al. (2019) even indicated that 32 allele-bearing individuals exhibit more beneficial values of kidney function parameters. Specifically, the wt/∆32 and ∆32/∆32 carriers exhibited a higher number of CD34+VEGFR2+ and CD34+VEGFR2+c-Kit+ cells than that in the wild type counterparts. This evidence concerns the gene CCR5 and kidney disorder.