Another study revealed that the endogenous MIP-1α promoter contains two consensus Runt-domain transcription factor (RUNX) sites in humans that are able to bind to an endogenous member of the mammalian family of RUNXs (RUNX1); this finding might indicate the possible pathogenesis of clonal hematopoiesis or myelodysplastic syndrome (MDS) upon marrow recovery [19]. The gene discussed is CCL3; the disease is myelodysplastic syndrome.