Also, miR-124a (which targets monocyte chemoattractant protein 1 (MCP-1) [143]), was found reduced in vivo in monocytes and neutrophils from APS and SLE patients, modulated in vitro by the autoantibodies from both autoimmune conditions, and related to the presence of a pathological increase of CIMT and to the occurrence of thrombotic events, as previously reported in other autoimmune conditions [33,220,221]. This evidence concerns the gene CCL2 and autoimmune polyendocrinopathy.