Consistent with our previous study,15 a study by Li et al also identified ASV could effectively regulate TGF‐β1 signalling to alleviate IPF.17 Considering that MTA1 status is a determinant of TGF‐β1‐induced EMT phenotypes,26 it is reasonably summing up that ASV regulated MTA1 expression in a TGF‐β1‐dependent way. This evidence concerns the gene TGFB1 and idiopathic pulmonary fibrosis.