TGFB1 and idiopathic pulmonary fibrosis: During EMT, the epithelial cells lose epithelial characteristics and acquired the mesenchymal phenotype with increased proliferative and migratory ability.3, 4, 5 It is well known that transforming growth factor‐beta1 (TGF‐β1), which is a pro‐fibrotic factor, has a pivotal role in inducing EMT.6 Therefore, novel therapeutic strategies should be focused on regulating TGF‐β1‐mediated EMT for effective management for IPF.