An et al. (2016) reported that in cervical carcinoma cells, α-Actinin-4 promotes EMT and tumorigenesis by regulating Snail expression and the Akt pathway. Pang et al. (2017) found that YB-1 promotes EMT and the progression of cervical carcinoma by upregulating Snail expression, which suggested that the YB-1/Snail/EMT axis may be used as a potential candidate for the diagnosis and therapy of cervical carcinoma metastasis. This evidence concerns the gene AKT1 and cervical carcinoma.