Alzheimer’s disease (AD), Parkinson’s disease (PD), and other amyloidosis-related diseases are induced when misfolded Amyloid β (Aβ), tau, and α-synuclein proteins aggregate in the brain or in other organs and when hyperphosphorylated tau aggregates in neurofibrillary tangles [5,6,7,8,9]. This evidence concerns the gene MAPT and early-onset autosomal dominant Alzheimer disease.