The activation of PI3K/AKT by FGFR1 was found to augment epithelial‐to‐mesenchymal transition and hence, metastasis in prostate cancer was shown in an earlier study.30 This led us to explore the role of miR‐3116 on FGFR1 and PI3K/AKT in the glioma cells under study. The gene discussed is FGFR1; the disease is prostate carcinoma.