Similarly, miR-29b has been linked to attenuating GBM angiogenesis and stemness by directly inhibiting B-cell lymphoma 2 homolog, Bcl2-like 2 (BCL2L2; also referred to as Bcl-w), which is amply expressed in the mesenchymal subtype of GBM [102]. Here, BCL2L2 is linked to glioblastoma.