CHRNA7 and pulmonary fibrosis: Perhaps, the activation of mAChRs, rather than nAChRs, in keratocytes and corneal myofibroblasts leads to an anti‐fibrotic response, rather than a pro‐fibrotic one, as it has been reported for airways that activation of α7 nAChR is involved in the progression of lung fibrosis.39, 40 Studies have shown contradicting results regarding the role of the mAChR antagonist atropine in fibrosis.