Altered gene expression of Sema3a, Trfr2 and Vldlr and altered protein levels of Foxp2. Embryonic day 18 mother infection led to significant gene alterations in frontal, hippocampal and cerebellar cortices of developing offspring. Significant atrophy in several brain areas and white matter thinning in corpus callosum. Altered levels of serotonin (P14, P35), 5-Hydroxyindoleacetic acid (P14) and taurine (P35). The gene discussed is VLDLR; the disease is infection.