In this regard, Gal1 KO pancreatic tumors have increased levels of CD3+, CD4+, and CD8+ T-effector cells together with decreased levels of CD11b+Gr1+ myeloid-derived suppressor cells, demonstrating that Gal1 overexpressed in PDA favors immune privilege [65]. The gene discussed is LGALS1; the disease is pancreatic neoplasm.