Thisanalysis supported the highly selective, though not exclusive, expression ofBMPR2, ACVRL1, and ENG on ECs, withmodestly increased expression of BMPR2 (1.52-fold), no changein ACVRL1 (1.0-fold), and upregulated expression ofENG (3.29-fold) by PAH compared to control ECs (Fig. 3). The gene discussed is ACVRL1; the disease is pulmonary arterial hypertension.