ARID1B and cancer: Additionally, the authors also suggest that the expression of ectonucleoside triphosphate phosphohydrolase‐7 (ENTPD7) or the inhibition of nucleotide synthesis in ARID1B‐depleted cells re‐establishes senescence, shedding more light on the novel mechanisms by which epigenetic regulators affect HCC progression, and suggesting that pro‐senescence therapies can be employed to treat SWI/SNF‐mutated cancers.