AKT1 and neoplasm: Other underlying mechanisms by which SMARCA4 promotes the proliferation of different tumour cells include stimulating the expression of MYC, inhibiting that of phosphatase and tensin homolog (PTEN), further reducing the active components in the phosphatidylinositol‐3 kinase‐protein kinase B (PI3K‐AKT) signalling pathway and promoting the function of the WNT signalling pathway.50, 51, 52