A follow‐up study elucidated the underlying mechanism in the AKI model: pFUS upregulates IFNγ in the injured kidney, which stimulates MSCs to produce IL‐10, an anti‐inflammatory cytokine that promotes recovery.107 They also demonstrated that IFNγ stimulation upregulates IL‐10 in MSCs in vitro and improves AKI outcomes more so than unstimulated MSCs. Here, IL10 is linked to acute kidney injury.