To verify that the IFNPS we identified is reflecting type I IFN– and not type II IFN–associated biology, we first measured the correlation between IFNPS and transcription of several components of IFN-γ–inducible gene signatures, IRF1, CXCL9, and SLAMF839,41, and found no correlation between the IFNPS and these genes in samples from patients with either SLE or myositis. The gene discussed is IFNG; the disease is systemic lupus erythematosus.