Moreover, NZ, alone and in association with doxorubicin, reduced the intratumor levels of ABCB1 (Figure 5C,D), potentially increasing the sensitivity to doxorubicin, increased the intratumor expression of ABCA1 (Figure 3C,D) and infiltration of Vγ9δ2 T-lymphocytes (Figure 3E), suggesting an enhanced recruitment of this set of immune cells within the tumor mass. The gene discussed is ABCA1; the disease is neoplasm.