Recently, the RET proto‐oncogene was shown to be upregulated in AML and might promote leukemogenesis through inhibition of mTORC1‐regulated autophagy (Rudat et al, 2018), stabilizing oncogenic proteins including FLT3/ITD that are susceptible to autophagic degradation. This evidence concerns the gene FLT3 and acute myeloid leukemia.