To identify the functional role of Nudt7 in lipid homeostasis, we analyzed lipid accumulation via modulation of Nudt7. Lipid accumulation and the number of BODIPY-positive cells (average of 0.2-fold with Nudt7 overexpression (Nudt7), 1.8-fold with Nudt7 knockdown (shNudt7) compared to KrasG12D CRC cells), were significantly increased in KrasG12D CRC cell lines, SNU-407, and SNU-C2A, and this increase was dramatically reduced by overexpression of Nudt7 (Figure S2). This evidence concerns the gene NUDT7 and colorectal carcinoma.