The literature consistently reassures the association between dyslipidemia and incidence of vascular events, with strong etiological contribution by increased triglyceride (TG), low-density lipoprotein (LDL), very-low-density lipoprotein (VLDL), and apolipoprotein B (apoB) levels, as well as by reduction of high-density lipoprotein (HDL) levels (4,5). This evidence concerns the gene APOB and metabolic syndrome.