Considering that we detected no impairments on nerve-specific electrophysiology parameters (Fig. 1G) and that we failed to find morphological evidence for a neuropathy in Dnm2 wt/K562E mice at 2 months and 1 year of age (Fig. 2 and Supplementary Material, Fig. S1), we reasoned that the CMAP defect may derive from a muscle phenotype in Dnm2 wt/K562E animals. Here, DNM2 is linked to neuropathy.