Repression of miRNA‐192 may promote collagen deposition in response to TGF‐β.68 During the last few years, various miRNAs have emerged as important members in the pathogenesis and progression of DKD through participation in fibrosis, inflammation, hypertrophy, autophagy, ER stress, oxidative stress, insulin resistance, and podocyte injury as summarized in Figure 2. Here, TGFB1 is linked to diabetic kidney disease.