However, it is at odds with previous reports, whereby upregulation of STMN1in breast cancer cell lines promote cell proliferation and carcinogenesis.53 This difference in abundance between CAB and CAO subjects could be due to cell or tissue type and specific to transformation.54 Not surprisingly, STMN1 was not detected in CAOAC subjects, suggesting its possible sensitivity toward the neoadjuvant drugs, paclitaxel55 and carboplatin. This evidence concerns the gene STMN1 and breast carcinoma.