To investigate whether the activation of the Wnt/β-catenin signaling pathway mediates the pro-proliferative roles of DANCR in cervical cancer, we treated DANCR stably overexpressed and control HeLa cells, and DANCR stably depleted and control C-33A cells with the canonical Wnt/β-catenin pathway inhibitor ICG-001. Here, DANCR is linked to cervical cancer.