The DDAHs association with FGF2 warrants further investigation as well, as DDAH2 has recently been shown to alleviate fibrosis associated with diabetic cardiomyopathy [38] and DDAH1 to inhibit the Wnt/β-catenin pathway, EMT, and gastric cancer cell migration [53]. This evidence concerns the gene DDAH1 and diabetic cardiomyopathy.