This classification was mainly dependent on the expression of linc-MD1 and LncMyod; the expression levels of linc-MD1 and LncMyod were largely increased in the disuse models, including the denervation, casting, and tail suspension models, but not in the systemically wasting models, including the dexamethasone-administration, cancer cachexia, and fasting models. The gene discussed is LINCMD1; the disease is cancer.