PI3 and infection: Furthermore, we showed that IAV post-transcriptionally inhibited the antimicrobial/anti-protease molecule elafin/trappin-2, which we have shown previously to be anti-inflammatory in a variety of settings (27–29) and protects the host against maladaptive neutrophilic inflammation in P.a (27, 28, 30) infections, as well as against Plasmodium-mediated lung damage (29).