Because both G-CSF and IFN-γ negatively correlated with HDAC inhibition in Mtb-infected Mφ1 and IFN-γ is known to play a major role in TB pathogenesis (70), we further explored the possible role of IFN-γ in Mφ1 that were differentiated in the presence of HDAC inhibitors (Figure S5A). The gene discussed is CSF3; the disease is tuberculosis.