Lastly, a group of molecules differentially expressed in H. pylori infected children lack sufficient evidence as to either their role in gastric damage in adults with H. pylori- induced gastritis or GC (BCL-2, LCN2, REG3A, CALCA, THBS1, CRABP1, PPAR-Y) or dysregulation in adulthood is contradictory with findings in children (miRNA146a, miRNA155, TIMP1). This evidence concerns the gene LCN2 and gastric cancer.