In addition, it promotes the autophagic degradation of EGFR in non-small cell lung cancer (NSCLC), inhibits growth and angiogenesis in prostate tumors by suppressing the protein kinase B (Akt)/mammalian target of rapamycin (mTOR)/P70S6K pathway (Pang et al., 2010; Xu et al., 2016). Here, AKT1 is linked to prostate neoplasm.