Of note, a recent study has provided a clear link between dysfunctional LRRK2 in dopamine neurons and altered glial response in Drosophila PD models, by showing that the genetic inhibition of the pro-inflammatory bone morphogenic protein (BMP) signaling in glia, was protective against PD-related neurotoxicity in aged mutant flies (Maksoud et al., 2019). The gene discussed is LRRK2; the disease is Parkinson disease.