Notably, cisplatin increased EphA2 and pS897/pY588, and BI‐D1870 blocked both the constitutive and cisplatin‐induced EphA2‐pS897 in the HGSC cells with high GPRC5A, concurrently increasing tumor‐suppressive EphA2‐pY588 (Fig 8C and D, Appendix Fig S7B). Here, CACNA1A is linked to neoplasm.