These phenotypes are however accompanied by paroxysmal dyskinesias [20], learning and memory deficits [21], and a variety of other behavioural deficits such as aggressivity and disturbances in sexual behaviour [44], supporting the much broader role of FGF14 in the brain as indicated by the heterogeneous presentation of clinical symptoms in SCA27 patients. This evidence concerns the gene FGF14 and paroxysmal dyskinesia.