These phenotypes are however accompanied by paroxysmal dyskinesias [20], learning and memory deficits [21], and a variety of other behavioural deficits such as aggressivity and disturbances in sexual behaviour [44], supporting the much broader role of FGF14 in the brain as indicated by the heterogeneous presentation of clinical symptoms in SCA27 patients. The gene discussed is FGF14; the disease is spinocerebellar ataxia type 27.