FKBP9 and glioblastoma: Consistent with these findings, we observed that both siRNA-mediated knockdown of ASK1 and exposure to NQDI-1 (ASK1 inhibitor) significantly impaired the increased clonogenic growth potential of FKBP9-overexpressing SF-539 and T98G cells (Fig. 5j and Additional file 3: Figure S3c), suggesting that ASK1 could play a role in the FKBP9-mediated effects on GBM cell growth.