MIR17 and rheumatoid arthritis: More recently, mi‐R17 has been claimed as a potential pathogenetic effector in RA, since it is more abundant in RA‐EVs than in those of healthy controls, and suppresses Treg induction by inhibiting the expression of TGF‐β II (TGFBR II) possibly contributing to the impairment of Treg homeostasis in RA patients.97