The regulation of BIM is partially mediated by the miR-17-92 cluster (also known as MIR17HG), in MYC-driven leukemia [74,75] and the inhibition of this specific microRNA endows leukemic cells with a pro-apoptotic phenotype [75], making microRNA networks an alternative entry point for interfering with MYC function in the B-cell precursor ALL. The gene discussed is MYC; the disease is acute lymphoblastic leukemia.