Later on, cases were reported to be associated with specific autoantibodies (autoimmune limbic encephalitis, ALE), either to cell membrane antigens like the N-methyl-D-aspartate receptor (NMDA-R) or the γ-aminobutyric acid receptor (GABA-R) [4], as well as to intracellular antigens like Hu (anti-neuronal nuclear antibody-1) in patients with lung cancer [5], Ma2 (paraneoplastic antigen Ma2) in patients with testicular tumors [6] and CRMP5/CV2 (collapsin response mediator protein 5) in patients with thymomas [7]. This evidence concerns the gene DPYSL5 and lung cancer.